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  Indian J Med Microbiol
 

Figure 6: Cell viability and inhibition of inflammatory cytokine NO, TNF-α, and PGE2 production on RAW 264.7 cells: Treated with ethyl acetate extract (a) 2.05 μg/mL, (b) 16.47 μg/mL, (c) 65.89 μg/mL and (d) Control (the arrows indicate cell dense zone). (e) Production of NO, (f) TNF-α, and (g) PGE2 which were pretreated with ethyl acetate extract# (20, 40, and 60 μg/mL) for 1 h followed by stimulation with 1 μg/mL of LPS for 24 h. Error bar represents mean ± standard deviation of triplicates, and marked with # are significantly different from control* at P < 0.05. NO: Nitric oxide; TNF-α: Tumor necrosis factor-alpha; PGE2: Prostaglandin E2, LPS: lipo-polysaccharide

Figure 6: Cell viability and inhibition of inflammatory cytokine NO, TNF-α, and PGE<sub>2</sub> production on RAW 264.7 cells: Treated with ethyl acetate extract (a) 2.05 μg/mL, (b) 16.47 μg/mL, (c) 65.89 μg/mL and (d) Control (the arrows indicate cell dense zone). (e) Production of NO, (f) TNF-α, and (g) PGE<sub>2</sub> which were pretreated with ethyl acetate extract<sup>#</sup> (20, 40, and 60 μg/mL) for 1 h followed by stimulation with 1 μg/mL of LPS for 24 h. Error bar represents mean ± standard deviation of triplicates, and marked with # are significantly different from control* at <i>P</i> < 0.05. NO: Nitric oxide; TNF-α: Tumor necrosis factor-alpha; PGE<sub>2</sub>: Prostaglandin E<sub>2</sub>, LPS: lipo-polysaccharide